Filtros : "FMRP" "Elias, Lucila Leico Kagohara" "Indexado no Scopus" Removido: "Oliveira, José Antonio Apparecido de" Limpar

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  • Source: Brain Research. Unidade: FMRP

    Subjects: EPILEPSIA DO LOBO TEMPORAL, HIPOTÁLAMO

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      UMEOKA, Eduardo Henrique de Lima et al. Functional characterization of the hypothalamic-pituitary-adrenal axis of the wistar audiogenic rat (WAR) strain. Brain Research, v. 1381, p. 141-147, 2011Tradução . . Acesso em: 30 abr. 2024.
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      Umeoka, E. H. de L., Garcia, S. B., Antunes-Rodrigues, J., Elias, L. L. K., & Garcia-Cairasco, N. (2011). Functional characterization of the hypothalamic-pituitary-adrenal axis of the wistar audiogenic rat (WAR) strain. Brain Research, 1381, 141-147.
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      Umeoka EH de L, Garcia SB, Antunes-Rodrigues J, Elias LLK, Garcia-Cairasco N. Functional characterization of the hypothalamic-pituitary-adrenal axis of the wistar audiogenic rat (WAR) strain. Brain Research. 2011 ; 1381 141-147.[citado 2024 abr. 30 ]
    • Vancouver

      Umeoka EH de L, Garcia SB, Antunes-Rodrigues J, Elias LLK, Garcia-Cairasco N. Functional characterization of the hypothalamic-pituitary-adrenal axis of the wistar audiogenic rat (WAR) strain. Brain Research. 2011 ; 1381 141-147.[citado 2024 abr. 30 ]
  • Source: Bioscience Reports. Unidade: FMRP

    Subjects: GASES INORGÂNICOS, ÓXIDO NÍTRICO, HIPOTÁLAMO, ENZIMAS

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      GOMES, Dayane Aparecida et al. Carbon monoxide and nitric oxide modulate hyperosmolality-induced oxitocin secretion by the hypothalamus in vitro. Bioscience Reports, v. 30, n. 5, p. 351-357, 2010Tradução . . Disponível em: https://doi.org/10.1042/bsr20090010. Acesso em: 30 abr. 2024.
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      Gomes, D. A., Paiva, A. G., Ventura, R. R., Elias, L. L. K., Cunha, F. de Q., & Antunes-Rodrigues, J. (2010). Carbon monoxide and nitric oxide modulate hyperosmolality-induced oxitocin secretion by the hypothalamus in vitro. Bioscience Reports, 30( 5), 351-357. doi:10.1042/bsr20090010
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      Gomes DA, Paiva AG, Ventura RR, Elias LLK, Cunha F de Q, Antunes-Rodrigues J. Carbon monoxide and nitric oxide modulate hyperosmolality-induced oxitocin secretion by the hypothalamus in vitro [Internet]. Bioscience Reports. 2010 ; 30( 5): 351-357.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/bsr20090010
    • Vancouver

      Gomes DA, Paiva AG, Ventura RR, Elias LLK, Cunha F de Q, Antunes-Rodrigues J. Carbon monoxide and nitric oxide modulate hyperosmolality-induced oxitocin secretion by the hypothalamus in vitro [Internet]. Bioscience Reports. 2010 ; 30( 5): 351-357.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/bsr20090010
  • Source: Autonomic Neuroscience: Basic and Clinical. Unidade: FMRP

    Subjects: ESTRESSE, SISTEMA NERVOSO CENTRAL, NEUROTRANSMISSORES

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      BUSNARDO, Cristiane et al. Paraventricular nucleus modulates autonomic and neuroendocrine responses to acute restraint stress in rats. Autonomic Neuroscience: Basic and Clinical, v. 158, n. 1/2, p. 51-57, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2010.06.003. Acesso em: 30 abr. 2024.
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      Busnardo, C., Tavares, R. F., Resstel, L. B. M., Elias, L. L. K., & Corrêa, F. M. de A. (2010). Paraventricular nucleus modulates autonomic and neuroendocrine responses to acute restraint stress in rats. Autonomic Neuroscience: Basic and Clinical, 158( 1/2), 51-57. doi:10.1016/j.autneu.2010.06.003
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      Busnardo C, Tavares RF, Resstel LBM, Elias LLK, Corrêa FM de A. Paraventricular nucleus modulates autonomic and neuroendocrine responses to acute restraint stress in rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2010 ; 158( 1/2): 51-57.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.autneu.2010.06.003
    • Vancouver

      Busnardo C, Tavares RF, Resstel LBM, Elias LLK, Corrêa FM de A. Paraventricular nucleus modulates autonomic and neuroendocrine responses to acute restraint stress in rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2010 ; 158( 1/2): 51-57.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.autneu.2010.06.003
  • Source: Hormones and Behavior. Unidade: FMRP

    Subjects: HORMÔNIOS GLICOCORTICOIDES, CONSUMO DE ALIMENTOS, HIPOTÁLAMO

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      UCHÔA, Ernane Torres et al. Corticotrophym-releasing factor mediates hypophagia after adrenalectomy, increasing meal-related satiety responses. Hormones and Behavior, v. 58, n. 5, p. 714-719, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2010.07.003. Acesso em: 30 abr. 2024.
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      Uchôa, E. T., Silva, L. E. C. M. da, Castro, M. de, Antunes-Rodrigues, J., & Elias, L. L. K. (2010). Corticotrophym-releasing factor mediates hypophagia after adrenalectomy, increasing meal-related satiety responses. Hormones and Behavior, 58( 5), 714-719. doi:10.1016/j.yhbeh.2010.07.003
    • NLM

      Uchôa ET, Silva LECM da, Castro M de, Antunes-Rodrigues J, Elias LLK. Corticotrophym-releasing factor mediates hypophagia after adrenalectomy, increasing meal-related satiety responses [Internet]. Hormones and Behavior. 2010 ; 58( 5): 714-719.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2010.07.003
    • Vancouver

      Uchôa ET, Silva LECM da, Castro M de, Antunes-Rodrigues J, Elias LLK. Corticotrophym-releasing factor mediates hypophagia after adrenalectomy, increasing meal-related satiety responses [Internet]. Hormones and Behavior. 2010 ; 58( 5): 714-719.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2010.07.003
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP

    Subjects: URINA (RENDIMENTO), OCITOCINA, SECREÇÃO (FISIOLOGIA)

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      MARGATHO, L. O e ELIAS, Lucila Leico Kagohara e ANTUNES-RODRIGUES, José. GABA in the central amygdaloid nucleus modulates the electrolyte excretion and hormonal responses to blood volume expansion in rats. Brazilian Journal of Medical and Biological Research, v. 42, n. 1, p. 114-121, 2009Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2009000100016. Acesso em: 30 abr. 2024.
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      Margatho, L. O., Elias, L. L. K., & Antunes-Rodrigues, J. (2009). GABA in the central amygdaloid nucleus modulates the electrolyte excretion and hormonal responses to blood volume expansion in rats. Brazilian Journal of Medical and Biological Research, 42( 1), 114-121. doi:10.1590/s0100-879x2009000100016
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      Margatho LO, Elias LLK, Antunes-Rodrigues J. GABA in the central amygdaloid nucleus modulates the electrolyte excretion and hormonal responses to blood volume expansion in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 1): 114-121.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/s0100-879x2009000100016
    • Vancouver

      Margatho LO, Elias LLK, Antunes-Rodrigues J. GABA in the central amygdaloid nucleus modulates the electrolyte excretion and hormonal responses to blood volume expansion in rats [Internet]. Brazilian Journal of Medical and Biological Research. 2009 ; 42( 1): 114-121.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/s0100-879x2009000100016
  • Source: Journal of Applied Physiology. Unidade: FMRP

    Subjects: HIPOTÁLAMO, ADRENALECTOMIA ANIMAL

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      UCHÔA, Ernane Torres et al. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses. Journal of Applied Physiology, v. 106, n. 2, p. 596-604, 2009Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.90865.2008. Acesso em: 30 abr. 2024.
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      Uchôa, E. T., Sabino, H. A. C., Ruginsk, S. G., Antunes-Rodrigues, J., & Elias, L. L. K. (2009). Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses. Journal of Applied Physiology, 106( 2), 596-604. doi:10.1152/japplphysiol.90865.2008
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      Uchôa ET, Sabino HAC, Ruginsk SG, Antunes-Rodrigues J, Elias LLK. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses [Internet]. Journal of Applied Physiology. 2009 ; 106( 2): 596-604.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/japplphysiol.90865.2008
    • Vancouver

      Uchôa ET, Sabino HAC, Ruginsk SG, Antunes-Rodrigues J, Elias LLK. Hypophagia induced by glucocorticoid deficiency is associated with an increased activation of satiety-related responses [Internet]. Journal of Applied Physiology. 2009 ; 106( 2): 596-604.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/japplphysiol.90865.2008
  • Source: Hormones and Behavior. Unidade: FMRP

    Subjects: OCITOCINA, HORMÔNIOS GLICOCORTICOIDES

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      UCHÔA, Ernane Torres et al. Hypothalamic oxytocin neurons modulate hypophagic effect induced by adrenalectomy. Hormones and Behavior, v. 56, n. 5, p. 532-538, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2009.09.007. Acesso em: 30 abr. 2024.
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      Uchôa, E. T., Silva, L. E. C. M. da, Castro, M. de, Antunes-Rodrigues, J., & Elias, L. L. K. (2009). Hypothalamic oxytocin neurons modulate hypophagic effect induced by adrenalectomy. Hormones and Behavior, 56( 5), 532-538. doi:10.1016/j.yhbeh.2009.09.007
    • NLM

      Uchôa ET, Silva LECM da, Castro M de, Antunes-Rodrigues J, Elias LLK. Hypothalamic oxytocin neurons modulate hypophagic effect induced by adrenalectomy [Internet]. Hormones and Behavior. 2009 ; 56( 5): 532-538.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2009.09.007
    • Vancouver

      Uchôa ET, Silva LECM da, Castro M de, Antunes-Rodrigues J, Elias LLK. Hypothalamic oxytocin neurons modulate hypophagic effect induced by adrenalectomy [Internet]. Hormones and Behavior. 2009 ; 56( 5): 532-538.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2009.09.007
  • Source: American Journal of Physiology : Regulatory, Integrative and Comparative Physiology. Unidade: FMRP

    Subjects: HIPOTÁLAMO, FISIOLOGIA CARDIOVASCULAR

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      BENEDETTI, Mauricio et al. Water deprivation increases Fos expression in hypothalamic corticotropin-releasing factor neurons induced by right atrial distension in awake rats. American Journal of Physiology : Regulatory, Integrative and Comparative Physiology, v. 295, n. 5, p. R1706-R1712, 2008Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00022.2008. Acesso em: 30 abr. 2024.
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      Benedetti, M., Rorato, R., Castro, M., Machado, B. H., Antunes-Rodrigues, J., & Elias, L. L. K. (2008). Water deprivation increases Fos expression in hypothalamic corticotropin-releasing factor neurons induced by right atrial distension in awake rats. American Journal of Physiology : Regulatory, Integrative and Comparative Physiology, 295( 5), R1706-R1712. doi:10.1152/ajpregu.00022.2008
    • NLM

      Benedetti M, Rorato R, Castro M, Machado BH, Antunes-Rodrigues J, Elias LLK. Water deprivation increases Fos expression in hypothalamic corticotropin-releasing factor neurons induced by right atrial distension in awake rats [Internet]. American Journal of Physiology : Regulatory, Integrative and Comparative Physiology. 2008 ; 295( 5): R1706-R1712.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpregu.00022.2008
    • Vancouver

      Benedetti M, Rorato R, Castro M, Machado BH, Antunes-Rodrigues J, Elias LLK. Water deprivation increases Fos expression in hypothalamic corticotropin-releasing factor neurons induced by right atrial distension in awake rats [Internet]. American Journal of Physiology : Regulatory, Integrative and Comparative Physiology. 2008 ; 295( 5): R1706-R1712.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpregu.00022.2008
  • Source: European Journal of Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, DOENÇAS METABÓLICAS

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      COLLARES, Cristhiana Viesti Advincula et al. Heterogeneity in the molecular basis of ACTH resistance syndrome. European Journal of Endocrinology, v. 159, n. 1, p. 61-68, 2008Tradução . . Disponível em: https://doi.org/10.1530/eje-08-0079. Acesso em: 30 abr. 2024.
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      Collares, C. V. A., Antunes-Rodrigues, J., Moreira, A. C., Franca, S. N., Pereira, L. A., Soares, M. M. S., et al. (2008). Heterogeneity in the molecular basis of ACTH resistance syndrome. European Journal of Endocrinology, 159( 1), 61-68. doi:10.1530/eje-08-0079
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      Collares CVA, Antunes-Rodrigues J, Moreira AC, Franca SN, Pereira LA, Soares MMS, Elias Júnior J, Clark AJ, Castro M de, Elias LLK. Heterogeneity in the molecular basis of ACTH resistance syndrome [Internet]. European Journal of Endocrinology. 2008 ; 159( 1): 61-68.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1530/eje-08-0079
    • Vancouver

      Collares CVA, Antunes-Rodrigues J, Moreira AC, Franca SN, Pereira LA, Soares MMS, Elias Júnior J, Clark AJ, Castro M de, Elias LLK. Heterogeneity in the molecular basis of ACTH resistance syndrome [Internet]. European Journal of Endocrinology. 2008 ; 159( 1): 61-68.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1530/eje-08-0079
  • Source: Hormone and Metabolic Research. Unidades: ICB, FMRP

    Subjects: ADRENALECTOMIA ANIMAL, FISIOLOGIA

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      GERMANO, C. M. R. et al. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research, v. 40, p. 842-847, 2008Tradução . . Acesso em: 30 abr. 2024.
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      Germano, C. M. R., Castro, M. de, Rorato, R., Costa, D. B., Antunes-Rodrigues, J., Elias, C. F., & Elias, L. L. K. (2008). Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research, 40, 842-847.
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      Germano CMR, Castro M de, Rorato R, Costa DB, Antunes-Rodrigues J, Elias CF, Elias LLK. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research. 2008 ; 40 842-847.[citado 2024 abr. 30 ]
    • Vancouver

      Germano CMR, Castro M de, Rorato R, Costa DB, Antunes-Rodrigues J, Elias CF, Elias LLK. Downregulation of melanocortin-4 receptor during refeeding and its modulation by adrenalectomy in rats. Hormone and Metabolic Research. 2008 ; 40 842-847.[citado 2024 abr. 30 ]
  • Source: Hormones and Behavior. Unidade: FMRP

    Subjects: NEUROENDOCRINOLOGIA, HIPOTÁLAMO, ADRENALECTOMIA ANIMAL

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      RORATO, R. et al. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons. Hormones and Behavior, v. 54, n. 1, p. 134-142, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2008.02.011. Acesso em: 30 abr. 2024.
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      Rorato, R., Borges, B. C., Benedetti, M., Germano, C. M. R., Antunes-Rodrigues, J., & Elias, L. L. K. (2008). Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons. Hormones and Behavior, 54( 1), 134-142. doi:10.1016/j.yhbeh.2008.02.011
    • NLM

      Rorato R, Borges BC, Benedetti M, Germano CMR, Antunes-Rodrigues J, Elias LLK. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons [Internet]. Hormones and Behavior. 2008 ; 54( 1): 134-142.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2008.02.011
    • Vancouver

      Rorato R, Borges BC, Benedetti M, Germano CMR, Antunes-Rodrigues J, Elias LLK. Adrenalectomy enhances endotoxemia-induced hypophagia: higher activation of corticotrophin-releasing-factor and proopiomelanocortin hypothalamic neurons [Internet]. Hormones and Behavior. 2008 ; 54( 1): 134-142.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2008.02.011
  • Source: Neuroscience. Unidade: FMRP

    Assunto: NEUROCIÊNCIAS

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      LAUAND, F. et al. Glucocorticoid modulation of atrial natriuretic peptide, oxytocin, vasopressin and Fos expression in response to osmotic, antiotensinergic and cholinergic stimulation. Neuroscience, v. 147, n. 1, p. 247-257, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2007.04.021. Acesso em: 30 abr. 2024.
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      Lauand, F., Ruginsk, S. G., Rodrigues, H. L. P., Reis, W. L., Castro, M. de, Elias, L. L. K., & Antunes-Rodrigues, J. (2007). Glucocorticoid modulation of atrial natriuretic peptide, oxytocin, vasopressin and Fos expression in response to osmotic, antiotensinergic and cholinergic stimulation. Neuroscience, 147( 1), 247-257. doi:10.1016/j.neuroscience.2007.04.021
    • NLM

      Lauand F, Ruginsk SG, Rodrigues HLP, Reis WL, Castro M de, Elias LLK, Antunes-Rodrigues J. Glucocorticoid modulation of atrial natriuretic peptide, oxytocin, vasopressin and Fos expression in response to osmotic, antiotensinergic and cholinergic stimulation [Internet]. Neuroscience. 2007 ; 147( 1): 247-257.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.04.021
    • Vancouver

      Lauand F, Ruginsk SG, Rodrigues HLP, Reis WL, Castro M de, Elias LLK, Antunes-Rodrigues J. Glucocorticoid modulation of atrial natriuretic peptide, oxytocin, vasopressin and Fos expression in response to osmotic, antiotensinergic and cholinergic stimulation [Internet]. Neuroscience. 2007 ; 147( 1): 247-257.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.neuroscience.2007.04.021
  • Source: Clinical Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, HIPOGONADISMO

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      VERSIANI, Beatriz R. et al. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism. Clinical Endocrinology, v. 66, p. 173-179, 2007Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2006.02702.x. Acesso em: 30 abr. 2024.
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      Versiani, B. R., Trarbach, E., Koenigkam-Santos, M., Santos, A. C. dos, Elias, L. L. K., Moreira, A. C., et al. (2007). Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism. Clinical Endocrinology, 66, 173-179. doi:10.1111/j.1365-2265.2006.02702.x
    • NLM

      Versiani BR, Trarbach E, Koenigkam-Santos M, Santos AC dos, Elias LLK, Moreira AC, Latronico AC, Castro M de. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism [Internet]. Clinical Endocrinology. 2007 ; 66 173-179.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02702.x
    • Vancouver

      Versiani BR, Trarbach E, Koenigkam-Santos M, Santos AC dos, Elias LLK, Moreira AC, Latronico AC, Castro M de. Clinical assessment and molecular analysis of GnRHR and KAL1 genes in males with idiopathic hypogonadotrophic hypogonadism [Internet]. Clinical Endocrinology. 2007 ; 66 173-179.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/j.1365-2265.2006.02702.x

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